High Blood Pressure

Worldwide, several hundred million people suffer from high blood pressure. Of all cardiovascular health conditions, this is the single largest epidemic. The epidemic spread of this disease is largely due to the fact that, until now, the causes of high blood pressure have been insufficiently, or not at all, understood.

Conventional medicine concedes that the causes of high blood pressure are unknown in over 90% of patients. The frequent medical diagnosis “essential hypertension” was established to describe high blood pressure conditions in which the causes remain unknown. Conventional, pharmaceutical-oriented medicine is confined to treating the symptoms of this disease. Beta-blockers, diuretics and other high blood pressure medications artificially lower the blood pressure (symptomoriented approach) without correcting the primary underlying problem — a “spasm” of the blood vessel wall.

Modern Cellular Medicine provides a breakthrough in our understanding of the causes, prevention and adjunct treatment of high blood pressure. The main cause of high blood pressure is a chronic deficiency of essential nutrients in millions of artery wall cells. Among other functions, these cells are responsible for the availability of “relaxing factors” (nitricoxide), which decrease vascular wall tension and keep the blood pressure in normal range. The natural amino acid arginine, vitamin C and other components of Dr. Rath’s Cellular Health recommendations contribute to the optimum availability of these artery wall relaxing factors. In contrast, chronic deficiency of these essential nutrients can result in spasms and a thickening of the blood vessel walls, which can eventually elevate blood pressure.

Featured Studies

Dr. Rath Research Institute

Fat Soluble vitamins affect composition of extracellular matrix deposited by human aortic smooth muscle and endothelial cells in vitro

Vitamin C inhibits the calcification process in human vascular smooth muscle cells

Beneficial metabolic effects of Vitamin D on arterial wall cells in vitro

Cardiovascular Effects of Cyclical Dietary Vitamin C Withdrawal in Mice Deficient in Internal Synthesis of Vitamin C and producing human lipoprotein (a): Gulo(-/-); Lp(a)+

Effects of Various Multi-Nutrient Supplements on the Production and Extracellular Deposition of Collagen I and IV by Human Aortic Smooth Muscle Cells

Hypoascorbemia induces atherosclerosis and vascular deposition of lipoprotein(a) in transgenic mice

Inhibition Of Collagen Synthesis By Select Calcium And Sodium Channel Blockers Can Be Mitigated By Ascorbic Acid And Ascorbyl Palmitate

A Nutrient Mixture Containing Ascorbic Acid, Lysine, Proline, Arginine, Cysteine, and Green Tea Extract Suppresses Autocrine Inflammatory Response in Cultured Human Aortic Smooth Muscle Cells

Bioflavonoids Effectively Inhibit Smooth Muscle Cell-Mediated Contraction of Collagen Matrix Induced by Angiotensin II

Enhancement of Cardio-Protective Effects and Attenuation of Adverse Effects of Female Sex Hormones on Cultured Human Vascular Smooth Muscle Cells by a Combination of Ascorbic Acid, Lysine, Proline, Arginine, Cysteine, and Epigallocatechin Gallate

Extracellular Matrix-Mediated Control of Aortic Smooth Muscle Growth and Migration by a Combination of Ascorbic Acid, Lysine, Proline, and Catechins

Anti-Atherogenic Effects of a Mixture of Ascorbic Acid, Lysine, Proline, Arginine, Cysteine, and Green Tea Phenolics in Human Aortic Smooth Muscle Cells

Plant-Derived Micronutrients Suppress Monocyte Ahesion to Cultured Human Aortic Endothelial Cell Layer by Modulating Its Extracellular Matrix Composition

Nutrient supplementation modulates angiotensin II-mediated atherosclerosis in ApoE KO mice

Evolution of angiotensin II-mediated atherosclerosis in ApoE KO mice

Clinical Studies

Further Scientific Studies

With Vitamin C

With other Micronutrients